APA (7th ed.) Citation

Vitiello, P. P., Cardone, C., Martini, G., Ciardiello, D., Belli, V., Matrone, N., . . . Martinelli, E. (2019). Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK pathway in KRAS-mutated human colorectal cancer cell lines. Journal of Experimental & Clinical Cancer Research (17569966), 38(1), N.PAG. https://doi.org/10.1186/s13046-019-1035-0

Chicago Style (17th ed.) Citation

Vitiello, Pietro Paolo, et al. "Receptor Tyrosine Kinase-dependent PI3K Activation Is an Escape Mechanism to Vertical Suppression of the EGFR/RAS/MAPK Pathway in KRAS-mutated Human Colorectal Cancer Cell Lines." Journal of Experimental & Clinical Cancer Research (17569966) 38, no. 1 (2019): N.PAG. https://doi.org/10.1186/s13046-019-1035-0.

MLA (9th ed.) Citation

Vitiello, Pietro Paolo, et al. "Receptor Tyrosine Kinase-dependent PI3K Activation Is an Escape Mechanism to Vertical Suppression of the EGFR/RAS/MAPK Pathway in KRAS-mutated Human Colorectal Cancer Cell Lines." Journal of Experimental & Clinical Cancer Research (17569966), vol. 38, no. 1, 2019, p. N.PAG, https://doi.org/10.1186/s13046-019-1035-0.