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Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK pathway in KRAS-mutated human colorectal cancer cell lines.

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Title: Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK pathway in KRAS-mutated human colorectal cancer cell lines.
Authors: Vitiello, Pietro Paolo1 pietropaolo.vitiello@gmail.com, Cardone, Claudia1 cardone.cla@gmail.com, Martini, Giulia1,2 giulia.mart14@gmail.com, Ciardiello, Davide1 davideciardiello@yahoo.it, Belli, Valentina1 valentina.belli@hotmail.com, Matrone, Nunzia1 nu.matrone@gmail.com, Barra, Giusi1 giusi_barra@hotmail.it, Napolitano, Stefania1,3 stenap@fastwebnet.it, Della Corte, Carmina1,3 carmy.dellacorte@gmail.com, Turano, Mimmo4 mimmo.turano@unina.it, Furia, Maria4 mfuria@unina.it, Troiani, Teresa1 teresa.troiani@unicampania.it, Morgillo, Floriana1 floriana.morgillo@unicampania.it, De Vita, Ferdinando1 ferdinando.devita@unicampania.it, Ciardiello, Fortunato1 fortunato.ciardiello@unicampania.it, Martinelli, Erika1 erika.martinelli@unicampania.it
Source: Journal of Experimental & Clinical Cancer Research (17569966). 1/28/2019, Vol. 38 Issue 1, pN.PAG-N.PAG. 1p.
Database: Academic Search Ultimate
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  Data: Receptor tyrosine kinase-dependent PI3K activation is an escape mechanism to vertical suppression of the EGFR/RAS/MAPK pathway in KRAS-mutated human colorectal cancer cell lines.
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