APA (7th ed.) Citation

Liu, Y. C. G., & Yen-Tung Teng, A. (2024). Distinct cross talk of IL-17 & TGF-β with the immature CD11c+TRAF6(-/-)-null myeloid dendritic cell-derived osteoclast precursor (mDDOCp) may engage signaling toward an alternative pathway of osteoclastogenesis for arthritic bone loss in vivo. Immunity, Inflammation & Disease, 12(2), 1. https://doi.org/10.1002/iid3.1173

Chicago Style (17th ed.) Citation

Liu, Yen Chun G., and Andy Yen-Tung Teng. "Distinct Cross Talk of IL-17 & TGF-β with the Immature CD11c+TRAF6(-/-)-null Myeloid Dendritic Cell-derived Osteoclast Precursor (mDDOCp) May Engage Signaling Toward an Alternative Pathway of Osteoclastogenesis for Arthritic Bone Loss in Vivo." Immunity, Inflammation & Disease 12, no. 2 (2024): 1. https://doi.org/10.1002/iid3.1173.

MLA (9th ed.) Citation

Liu, Yen Chun G., and Andy Yen-Tung Teng. "Distinct Cross Talk of IL-17 & TGF-β with the Immature CD11c+TRAF6(-/-)-null Myeloid Dendritic Cell-derived Osteoclast Precursor (mDDOCp) May Engage Signaling Toward an Alternative Pathway of Osteoclastogenesis for Arthritic Bone Loss in Vivo." Immunity, Inflammation & Disease, vol. 12, no. 2, 2024, p. 1, https://doi.org/10.1002/iid3.1173.