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IκBε is a key regulator of B cell expansion by providing negative feedback on cRel and RelA in a stimulus-specific manner.

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Title: IκBε is a key regulator of B cell expansion by providing negative feedback on cRel and RelA in a stimulus-specific manner.
Authors: Alves BN; Signaling Systems Laboratory, Department of Chemistry and Biochemistry, University of California, San Diego, La Jolla, CA 92093;, Tsui R, Almaden J, Shokhirev MN, Davis-Turak J, Fujimoto J, Birnbaum H, Ponomarenko J, Hoffmann A
Source: Journal of immunology (Baltimore, Md. : 1950) [J Immunol] 2014 Apr 01; Vol. 192 (7), pp. 3121-32. Date of Electronic Publication: 2014 Mar 03.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
Journal Info: Publisher: Oxford University Press Country of Publication: England NLM ID: 2985117R Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1550-6606 (Electronic) Linking ISSN: 00221767 NLM ISO Abbreviation: J Immunol Subsets: MEDLINE
Database: MEDLINE Ultimate
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An: 24591377
AccessLevel: 2
PubType: Academic Journal
PubTypeId: academicJournal
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  Data: IκBε is a key regulator of B cell expansion by providing negative feedback on cRel and RelA in a stimulus-specific manner.
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  Data: <searchLink fieldCode="AU" term="%22Alves+BN%22">Alves BN</searchLink>; Signaling Systems Laboratory, Department of Chemistry and Biochemistry, University of California, San Diego, La Jolla, CA 92093;<br /><searchLink fieldCode="AU" term="%22Tsui+R%22">Tsui R</searchLink><br /><searchLink fieldCode="AU" term="%22Almaden+J%22">Almaden J</searchLink><br /><searchLink fieldCode="AU" term="%22Shokhirev+MN%22">Shokhirev MN</searchLink><br /><searchLink fieldCode="AU" term="%22Davis-Turak+J%22">Davis-Turak J</searchLink><br /><searchLink fieldCode="AU" term="%22Fujimoto+J%22">Fujimoto J</searchLink><br /><searchLink fieldCode="AU" term="%22Birnbaum+H%22">Birnbaum H</searchLink><br /><searchLink fieldCode="AU" term="%22Ponomarenko+J%22">Ponomarenko J</searchLink><br /><searchLink fieldCode="AU" term="%22Hoffmann+A%22">Hoffmann A</searchLink>
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  Data: <searchLink fieldCode="JN" term="%222985117R%22">Journal of immunology (Baltimore, Md. : 1950)</searchLink> [J Immunol] 2014 Apr 01; Vol. 192 (7), pp. 3121-32. <i>Date of Electronic Publication: </i>2014 Mar 03.
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  Data: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Oxford+University+Press%22">Oxford University Press </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>2985117R <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1550-6606 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200221767%22">00221767 </searchLink><i>NLM ISO Abbreviation: </i>J Immunol <i>Subsets: </i>MEDLINE
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        Value: 10.4049/jimmunol.1302351
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      – Code: eng
        Text: English
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      – TitleFull: IκBε is a key regulator of B cell expansion by providing negative feedback on cRel and RelA in a stimulus-specific manner.
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              Text: 2014 Apr 01
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              Y: 2014
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