Attenuated IGF-1 predicts all-cause and cardiovascular mortality in a Black population: A five-year prospective study.

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Bibliographic Details
Title: Attenuated IGF-1 predicts all-cause and cardiovascular mortality in a Black population: A five-year prospective study.
Authors: Schutte AE; Hypertension in Africa Research Team (HART), North-West University, Potchefstroom, South Africa Medical Research Council, Unit for Hypertension and Cardiovascular Disease, North-West University, Potchefstroom, South Africa alta.schutte@nwu.ac.za., Conti E; Department of Clinical and Molecular Medicine, University of Rome, Sapienza, Italy., Mels CM; Hypertension in Africa Research Team (HART), North-West University, Potchefstroom, South Africa., Smith W; Hypertension in Africa Research Team (HART), North-West University, Potchefstroom, South Africa., Kruger R; Hypertension in Africa Research Team (HART), North-West University, Potchefstroom, South Africa., Botha S; Hypertension in Africa Research Team (HART), North-West University, Potchefstroom, South Africa., Gnessi L; Department of Experimental Medicine, Pathophysiology and Endocrinology Unit, University of Rome, Sapienza, Italy., Volpe M; Department of Clinical and Molecular Medicine, University of Rome, Sapienza, Italy IRCCS Neuromed, Pozzilli, Italy., Huisman HW; Hypertension in Africa Research Team (HART), North-West University, Potchefstroom, South Africa Medical Research Council, Unit for Hypertension and Cardiovascular Disease, North-West University, Potchefstroom, South Africa.
Source: European journal of preventive cardiology [Eur J Prev Cardiol] 2016 Nov; Vol. 23 (16), pp. 1690-1699. Date of Electronic Publication: 2016 Jul 22.
Publication Type: Journal Article; Multicenter Study
Journal Info: Publisher: Oxford University Press Country of Publication: England NLM ID: 101564430 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2047-4881 (Electronic) Linking ISSN: 20474873 NLM ISO Abbreviation: Eur J Prev Cardiol Subsets: MEDLINE
Database: MEDLINE Ultimate
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