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A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses.

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Title: A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses.
Authors: Bent EH; The David H. Koch Institute for Integrative Cancer Research, Cambridge, Massachusetts 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA., Gilbert LA; The David H. Koch Institute for Integrative Cancer Research, Cambridge, Massachusetts 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA., Hemann MT; The David H. Koch Institute for Integrative Cancer Research, Cambridge, Massachusetts 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA.
Source: Genes & development [Genes Dev] 2016 Aug 15; Vol. 30 (16), pp. 1811-21. Date of Electronic Publication: 2016 Aug 26.
Publication Type: Journal Article
Journal Info: Publisher: Cold Spring Harbor Laboratory Press Country of Publication: United States NLM ID: 8711660 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1549-5477 (Electronic) Linking ISSN: 08909369 NLM ISO Abbreviation: Genes Dev Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses.
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  Data: <searchLink fieldCode="AU" term="%22Bent+EH%22">Bent EH</searchLink>; The David H. Koch Institute for Integrative Cancer Research, Cambridge, Massachusetts 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA.<br /><searchLink fieldCode="AU" term="%22Gilbert+LA%22">Gilbert LA</searchLink>; The David H. Koch Institute for Integrative Cancer Research, Cambridge, Massachusetts 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA.<br /><searchLink fieldCode="AU" term="%22Hemann+MT%22">Hemann MT</searchLink>; The David H. Koch Institute for Integrative Cancer Research, Cambridge, Massachusetts 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, Massachusetts 02139, USA.
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  Data: <searchLink fieldCode="JN" term="%228711660%22">Genes & development</searchLink> [Genes Dev] 2016 Aug 15; Vol. 30 (16), pp. 1811-21. <i>Date of Electronic Publication: </i>2016 Aug 26.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Cold+Spring+Harbor+Laboratory+Press%22">Cold Spring Harbor Laboratory Press </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>8711660 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1549-5477 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2208909369%22">08909369 </searchLink><i>NLM ISO Abbreviation: </i>Genes Dev <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=27566778
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        Value: 10.1101/gad.284851.116
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      – Code: eng
        Text: English
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        StartPage: 1811
    Titles:
      – TitleFull: A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses.
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            NameFull: Bent EH
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            NameFull: Gilbert LA
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            – D: 15
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              Text: 2016 Aug 15
              Type: published
              Y: 2016
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              Value: 30
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              Value: 16
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