APA (7th ed.) Citation

J, T., RS, P., LG, P., RDR, O., KA, L., PB, D., . . . F, d. Q. C. (2018). Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis research & therapy, 20(1), 119. https://doi.org/10.1186/s13075-018-1609-9

Chicago Style (17th ed.) Citation

J, Talbot, et al. "Smoking-induced Aggravation of Experimental Arthritis Is Dependent of Aryl Hydrocarbon Receptor Activation in Th17 Cells." Arthritis Research & Therapy 20, no. 1 (2018): 119. https://doi.org/10.1186/s13075-018-1609-9.

MLA (9th ed.) Citation

J, Talbot, et al. "Smoking-induced Aggravation of Experimental Arthritis Is Dependent of Aryl Hydrocarbon Receptor Activation in Th17 Cells." Arthritis Research & Therapy, vol. 20, no. 1, 2018, p. 119, https://doi.org/10.1186/s13075-018-1609-9.