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Retrograde axonal transport of rabies virus is unaffected by interferon treatment but blocked by emetine locally in axons.

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Title: Retrograde axonal transport of rabies virus is unaffected by interferon treatment but blocked by emetine locally in axons.
Authors: MacGibeny MA; Department of Molecular Biology and Princeton Neuroscience Institute, Princeton University, Princeton, New Jersey, United States of America., Koyuncu OO; Department of Molecular Biology and Princeton Neuroscience Institute, Princeton University, Princeton, New Jersey, United States of America., Wirblich C; Department of Microbiology and Immunology, Thomas Jefferson University, Philadelphia, Pennsylvania, United States of America., Schnell MJ; Department of Microbiology and Immunology, Thomas Jefferson University, Philadelphia, Pennsylvania, United States of America., Enquist LW; Department of Molecular Biology and Princeton Neuroscience Institute, Princeton University, Princeton, New Jersey, United States of America.
Source: PLoS pathogens [PLoS Pathog] 2018 Jul 20; Vol. 14 (7), pp. e1007188. Date of Electronic Publication: 2018 Jul 20 (Print Publication: 2018).
Publication Type: Journal Article; Research Support, N.I.H., Extramural
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Retrograde axonal transport of rabies virus is unaffected by interferon treatment but blocked by emetine locally in axons.
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  Data: <searchLink fieldCode="AU" term="%22MacGibeny+MA%22">MacGibeny MA</searchLink>; Department of Molecular Biology and Princeton Neuroscience Institute, Princeton University, Princeton, New Jersey, United States of America.<br /><searchLink fieldCode="AU" term="%22Koyuncu+OO%22">Koyuncu OO</searchLink>; Department of Molecular Biology and Princeton Neuroscience Institute, Princeton University, Princeton, New Jersey, United States of America.<br /><searchLink fieldCode="AU" term="%22Wirblich+C%22">Wirblich C</searchLink>; Department of Microbiology and Immunology, Thomas Jefferson University, Philadelphia, Pennsylvania, United States of America.<br /><searchLink fieldCode="AU" term="%22Schnell+MJ%22">Schnell MJ</searchLink>; Department of Microbiology and Immunology, Thomas Jefferson University, Philadelphia, Pennsylvania, United States of America.<br /><searchLink fieldCode="AU" term="%22Enquist+LW%22">Enquist LW</searchLink>; Department of Molecular Biology and Princeton Neuroscience Institute, Princeton University, Princeton, New Jersey, United States of America.
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  Data: <searchLink fieldCode="JN" term="%22101238921%22">PLoS pathogens</searchLink> [PLoS Pathog] 2018 Jul 20; Vol. 14 (7), pp. e1007188. <i>Date of Electronic Publication: </i>2018 Jul 20 (<i>Print Publication: </i>2018).
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Public+Library+of+Science%22">Public Library of Science </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>101238921 <i>Publication Model: </i>eCollection <i>Cited Medium: </i>Internet <i>ISSN: </i>1553-7374 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2215537366%22">15537366 </searchLink><i>NLM ISO Abbreviation: </i>PLoS Pathog <i>Subsets: </i>MEDLINE
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        Value: 10.1371/journal.ppat.1007188
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        Text: English
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              Text: 2018 Jul 20
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