PHF1 fusions cause distinct gene expression and chromatin accessibility profiles in ossifying fibromyxoid tumors and mesenchymal cells.

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Title: PHF1 fusions cause distinct gene expression and chromatin accessibility profiles in ossifying fibromyxoid tumors and mesenchymal cells.
Authors: Hofvander J; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden., Jo VY; Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA., Fletcher CDM; Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA., Puls F; Department of Clinical Pathology and Genetics, Sahlgrenska University Hospital, Gothenburg, Sweden., Flucke U; Department of Pathology, Radboud University Medical Centre, Nijmegen, The Netherlands., Nilsson J; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden., Magnusson L; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden., Mertens F; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden. Fredrik.mertens@med.lu.se.; Department of Clinical Genetics and Pathology, Skåne University Hospital, SE-221 85, Lund, Sweden. Fredrik.mertens@med.lu.se.
Source: Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc [Mod Pathol] 2020 Jul; Vol. 33 (7), pp. 1331-1340. Date of Electronic Publication: 2020 Jan 13.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Elsevier Inc Country of Publication: United States NLM ID: 8806605 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1530-0285 (Electronic) Linking ISSN: 08933952 NLM ISO Abbreviation: Mod Pathol Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: PHF1 fusions cause distinct gene expression and chromatin accessibility profiles in ossifying fibromyxoid tumors and mesenchymal cells.
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  Data: <searchLink fieldCode="AU" term="%22Hofvander+J%22">Hofvander J</searchLink>; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Jo+VY%22">Jo VY</searchLink>; Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.<br /><searchLink fieldCode="AU" term="%22Fletcher+CDM%22">Fletcher CDM</searchLink>; Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.<br /><searchLink fieldCode="AU" term="%22Puls+F%22">Puls F</searchLink>; Department of Clinical Pathology and Genetics, Sahlgrenska University Hospital, Gothenburg, Sweden.<br /><searchLink fieldCode="AU" term="%22Flucke+U%22">Flucke U</searchLink>; Department of Pathology, Radboud University Medical Centre, Nijmegen, The Netherlands.<br /><searchLink fieldCode="AU" term="%22Nilsson+J%22">Nilsson J</searchLink>; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Magnusson+L%22">Magnusson L</searchLink>; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden.<br /><searchLink fieldCode="AU" term="%22Mertens+F%22">Mertens F</searchLink>; Division of Clinical Genetics, Department of Laboratory Medicine, Lund University, Lund, Sweden. Fredrik.mertens@med.lu.se.; Department of Clinical Genetics and Pathology, Skåne University Hospital, SE-221 85, Lund, Sweden. Fredrik.mertens@med.lu.se.
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  Data: <searchLink fieldCode="JN" term="%228806605%22">Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc</searchLink> [Mod Pathol] 2020 Jul; Vol. 33 (7), pp. 1331-1340. <i>Date of Electronic Publication: </i>2020 Jan 13.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier+Inc%22">Elsevier Inc </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>8806605 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1530-0285 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2208933952%22">08933952 </searchLink><i>NLM ISO Abbreviation: </i>Mod Pathol <i>Subsets: </i>MEDLINE
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        Value: 10.1038/s41379-020-0457-8
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        Text: English
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        StartPage: 1331
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      – TitleFull: PHF1 fusions cause distinct gene expression and chromatin accessibility profiles in ossifying fibromyxoid tumors and mesenchymal cells.
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              Text: 2020 Jul
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              Y: 2020
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