Cytoplasmic zinc promotes IL-1β production by monocytes and macrophages through mTORC1-induced glycolysis in rheumatoid arthritis.

Saved in:
Bibliographic Details
Title: Cytoplasmic zinc promotes IL-1β production by monocytes and macrophages through mTORC1-induced glycolysis in rheumatoid arthritis.
Authors: Kim B; Laboratory of Autoimmunity and Inflammation (LAI), Department of Biomedical Sciences, and BK21Plus Biomedical Science Project, Seoul National University College of Medicine, Seoul 03080, Republic of Korea., Kim HY; Laboratory of Autoimmunity and Inflammation (LAI), Department of Biomedical Sciences, and BK21Plus Biomedical Science Project, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Department of Microbiology and Immunology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Institute of Infectious Diseases, Seoul National University College of Medicine, Seoul 03080, Republic of Korea., Yoon BR; Department of Microbiology and Immunology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea., Yeo J; Division of Rheumatology, Department of Internal Medicine, Seoul National University College of Medicine, Seoul 03080, Republic of Korea., In Jung J; Division of Rheumatology, Department of Internal Medicine, Seoul National University College of Medicine, Seoul 03080, Republic of Korea., Yu KS; Department of Clinical Pharmacology and Therapeutics, Seoul National University College of Medicine and Hospital, Seoul 03080, Republic of Korea., Kim HC; Department of Preventive Medicine, Yonsei University College of Medicine, Seoul 03722, Republic of Korea., Yoo SJ; Department of Internal Medicine, Chungnam National University School of Medicine, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Republic of Korea., Park JK; Division of Rheumatology, Department of Internal Medicine, Seoul National University College of Medicine, Seoul 03080, Republic of Korea., Kang SW; Department of Internal Medicine, Chungnam National University School of Medicine, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Republic of Korea., Lee WW; Laboratory of Autoimmunity and Inflammation (LAI), Department of Biomedical Sciences, and BK21Plus Biomedical Science Project, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Department of Microbiology and Immunology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Cancer Research Institute, Ischemic/Hypoxic Disease Institute, and Institute of Infectious Diseases, Seoul National University College of Medicine, Seoul National University Hospital Biomedical Research Institute, Seoul 03080, Republic of Korea.
Source: Science signaling [Sci Signal] 2022 Jan 11; Vol. 15 (716), pp. eabi7400. Date of Electronic Publication: 2022 Jan 11.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Association for the Advancement of Science Country of Publication: United States NLM ID: 101465400 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1937-9145 (Electronic) Linking ISSN: 19450877 NLM ISO Abbreviation: Sci Signal Subsets: MEDLINE
Database: MEDLINE Ultimate
Full text is not displayed to guests.
FullText Links:
  – Type: pdflink
Text:
  Availability: 1
Header DbId: mdl
DbLabel: MEDLINE Ultimate
An: 35015571
AccessLevel: 2
PubType: Academic Journal
PubTypeId: academicJournal
PreciseRelevancyScore: 0
IllustrationInfo
Items – Name: Title
  Label: Title
  Group: Ti
  Data: Cytoplasmic zinc promotes IL-1β production by monocytes and macrophages through mTORC1-induced glycolysis in rheumatoid arthritis.
– Name: Author
  Label: Authors
  Group: Au
  Data: <searchLink fieldCode="AU" term="%22Kim+B%22">Kim B</searchLink>; Laboratory of Autoimmunity and Inflammation (LAI), Department of Biomedical Sciences, and BK21Plus Biomedical Science Project, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kim+HY%22">Kim HY</searchLink>; Laboratory of Autoimmunity and Inflammation (LAI), Department of Biomedical Sciences, and BK21Plus Biomedical Science Project, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Department of Microbiology and Immunology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Institute of Infectious Diseases, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Yoon+BR%22">Yoon BR</searchLink>; Department of Microbiology and Immunology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Yeo+J%22">Yeo J</searchLink>; Division of Rheumatology, Department of Internal Medicine, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22In+Jung+J%22">In Jung J</searchLink>; Division of Rheumatology, Department of Internal Medicine, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Yu+KS%22">Yu KS</searchLink>; Department of Clinical Pharmacology and Therapeutics, Seoul National University College of Medicine and Hospital, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kim+HC%22">Kim HC</searchLink>; Department of Preventive Medicine, Yonsei University College of Medicine, Seoul 03722, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Yoo+SJ%22">Yoo SJ</searchLink>; Department of Internal Medicine, Chungnam National University School of Medicine, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Park+JK%22">Park JK</searchLink>; Division of Rheumatology, Department of Internal Medicine, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Kang+SW%22">Kang SW</searchLink>; Department of Internal Medicine, Chungnam National University School of Medicine, 282 Munhwa-ro, Jung-gu, Daejeon 35015, Republic of Korea.<br /><searchLink fieldCode="AU" term="%22Lee+WW%22">Lee WW</searchLink>; Laboratory of Autoimmunity and Inflammation (LAI), Department of Biomedical Sciences, and BK21Plus Biomedical Science Project, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Department of Microbiology and Immunology, Seoul National University College of Medicine, Seoul 03080, Republic of Korea.; Cancer Research Institute, Ischemic/Hypoxic Disease Institute, and Institute of Infectious Diseases, Seoul National University College of Medicine, Seoul National University Hospital Biomedical Research Institute, Seoul 03080, Republic of Korea.
– Name: TitleSource
  Label: Source
  Group: Src
  Data: <searchLink fieldCode="JN" term="%22101465400%22">Science signaling</searchLink> [Sci Signal] 2022 Jan 11; Vol. 15 (716), pp. eabi7400. <i>Date of Electronic Publication: </i>2022 Jan 11.
– Name: TypePub
  Label: Publication Type
  Group: TypPub
  Data: Journal Article; Research Support, Non-U.S. Gov't
– Name: TitleSource
  Label: Journal Info
  Group: Src
  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22American+Association+for+the+Advancement+of+Science%22">American Association for the Advancement of Science </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>101465400 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1937-9145 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2219450877%22">19450877 </searchLink><i>NLM ISO Abbreviation: </i>Sci Signal <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=35015571
RecordInfo BibRecord:
  BibEntity:
    Identifiers:
      – Type: doi
        Value: 10.1126/scisignal.abi7400
    Languages:
      – Code: eng
        Text: English
    PhysicalDescription:
      Pagination:
        StartPage: eabi7400
    Titles:
      – TitleFull: Cytoplasmic zinc promotes IL-1β production by monocytes and macrophages through mTORC1-induced glycolysis in rheumatoid arthritis.
        Type: main
  BibRelationships:
    HasContributorRelationships:
      – PersonEntity:
          Name:
            NameFull: Kim B
      – PersonEntity:
          Name:
            NameFull: Kim HY
      – PersonEntity:
          Name:
            NameFull: Yoon BR
      – PersonEntity:
          Name:
            NameFull: Yeo J
      – PersonEntity:
          Name:
            NameFull: In Jung J
      – PersonEntity:
          Name:
            NameFull: Yu KS
      – PersonEntity:
          Name:
            NameFull: Kim HC
      – PersonEntity:
          Name:
            NameFull: Yoo SJ
      – PersonEntity:
          Name:
            NameFull: Park JK
      – PersonEntity:
          Name:
            NameFull: Kang SW
      – PersonEntity:
          Name:
            NameFull: Lee WW
    IsPartOfRelationships:
      – BibEntity:
          Dates:
            – D: 11
              M: 01
              Text: 2022 Jan 11
              Type: published
              Y: 2022
          Identifiers:
            – Type: issn-electronic
              Value: 1937-9145
          Numbering:
            – Type: volume
              Value: 15
            – Type: issue
              Value: 716
          Titles:
            – TitleFull: Science signaling
              Type: main
ResultId 1