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Blood vessel occlusion by Cryptococcus neoformans is a mechanism for haemorrhagic dissemination of infection.

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Title: Blood vessel occlusion by Cryptococcus neoformans is a mechanism for haemorrhagic dissemination of infection.
Authors: Gibson JF; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.; Institute of Molecular and Cell Biology, Agency of Science, Technology and Research (A-Star), Singapore., Bojarczuk A; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.; Faculty of Physical Education, Gdansk University of Physical Education and Sport, Gdansk, Poland., Evans RJ; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom., Kamuyango AA; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom., Hotham R; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom., Lagendijk AK; Division of Genomics of Development and Disease, Institute for Molecular Bioscience, University of Queensland, Brisbane, Australia., Hogan BM; Division of Genomics of Development and Disease, Institute for Molecular Bioscience, University of Queensland, Brisbane, Australia., Ingham PW; Institute of Molecular and Cell Biology, Agency of Science, Technology and Research (A-Star), Singapore.; Lee Kong Chian School of Medicine, Nanyang Technological University, Singapore., Renshaw SA; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom., Johnston SA; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.
Source: PLoS pathogens [PLoS Pathog] 2022 Apr 21; Vol. 18 (4), pp. e1010389. Date of Electronic Publication: 2022 Apr 21 (Print Publication: 2022).
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Blood vessel occlusion by Cryptococcus neoformans is a mechanism for haemorrhagic dissemination of infection.
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  Data: <searchLink fieldCode="AU" term="%22Gibson+JF%22">Gibson JF</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.; Institute of Molecular and Cell Biology, Agency of Science, Technology and Research (A-Star), Singapore.<br /><searchLink fieldCode="AU" term="%22Bojarczuk+A%22">Bojarczuk A</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.; Faculty of Physical Education, Gdansk University of Physical Education and Sport, Gdansk, Poland.<br /><searchLink fieldCode="AU" term="%22Evans+RJ%22">Evans RJ</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.<br /><searchLink fieldCode="AU" term="%22Kamuyango+AA%22">Kamuyango AA</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.<br /><searchLink fieldCode="AU" term="%22Hotham+R%22">Hotham R</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.<br /><searchLink fieldCode="AU" term="%22Lagendijk+AK%22">Lagendijk AK</searchLink>; Division of Genomics of Development and Disease, Institute for Molecular Bioscience, University of Queensland, Brisbane, Australia.<br /><searchLink fieldCode="AU" term="%22Hogan+BM%22">Hogan BM</searchLink>; Division of Genomics of Development and Disease, Institute for Molecular Bioscience, University of Queensland, Brisbane, Australia.<br /><searchLink fieldCode="AU" term="%22Ingham+PW%22">Ingham PW</searchLink>; Institute of Molecular and Cell Biology, Agency of Science, Technology and Research (A-Star), Singapore.; Lee Kong Chian School of Medicine, Nanyang Technological University, Singapore.<br /><searchLink fieldCode="AU" term="%22Renshaw+SA%22">Renshaw SA</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.<br /><searchLink fieldCode="AU" term="%22Johnston+SA%22">Johnston SA</searchLink>; Department of Infection, Immunity and Cardiovascular disease, Bateson Centre and Florey Institute, University of Sheffield, United Kingdom.
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  Data: <searchLink fieldCode="JN" term="%22101238921%22">PLoS pathogens</searchLink> [PLoS Pathog] 2022 Apr 21; Vol. 18 (4), pp. e1010389. <i>Date of Electronic Publication: </i>2022 Apr 21 (<i>Print Publication: </i>2022).
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              Text: 2022 Apr 21
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