Deletion of skeletal muscle Akt1/2 causes osteosarcopenia and reduces lifespan in mice.
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| Title: | Deletion of skeletal muscle Akt1/2 causes osteosarcopenia and reduces lifespan in mice. |
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| Authors: | Sasako T; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.; Department of Molecular Diabetic Medicine, Diabetes Research Center, National Center for Global Health and Medicine, Tokyo, Japan., Umehara T; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan., Soeda K; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.; Department of Molecular Diabetic Medicine, Diabetes Research Center, National Center for Global Health and Medicine, Tokyo, Japan., Kaneko K; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan., Suzuki M; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan., Kobayashi N; Department of Molecular Diabetic Medicine, Diabetes Research Center, National Center for Global Health and Medicine, Tokyo, Japan., Okazaki Y; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan., Tamura-Nakano M; Communal Laboratory, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan., Chiba T; Department of Systems BioMedicine, Tokyo Medical and Dental University, Tokyo, Japan., Accili D; Columbia University College of Physicians & Surgeons, Department of Medicine, New York, NY, USA., Kahn CR; Joslin Diabetes Center, Harvard Medical School, Boston, MA, USA., Noda T; Department of Cell Biology, Cancer Institute, Japanese Foundation of Cancer Research, Tokyo, Japan., Asahara H; Department of Systems BioMedicine, Tokyo Medical and Dental University, Tokyo, Japan., Yamauchi T; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan., Kadowaki T; Department of Diabetes and Metabolic Diseases, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan. t-kadowaki@toranomon.kkr.or.jp.; Toranomon Hospital, Tokyo, Japan. t-kadowaki@toranomon.kkr.or.jp., Ueki K; Department of Molecular Diabetic Medicine, Diabetes Research Center, National Center for Global Health and Medicine, Tokyo, Japan. uekik@ri.ncgm.go.jp.; Department of Molecular Diabetetology, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan. uekik@ri.ncgm.go.jp. |
| Source: | Nature communications [Nat Commun] 2022 Oct 05; Vol. 13 (1), pp. 5655. Date of Electronic Publication: 2022 Oct 05. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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