Overexpression of miR-29a and miR-29b is involved in imatinib resistance via abrogated NF1 expression and increased ERK1/2 activation in chronic myeloid leukemia cells.
Saved in:
| Title: | Overexpression of miR-29a and miR-29b is involved in imatinib resistance via abrogated NF1 expression and increased ERK1/2 activation in chronic myeloid leukemia cells. |
|---|---|
| Authors: | Tsubaki M; Laboratory of Pharmacotherapy, Faculty of Pharmaceutical Sciences at Kagawa Campus, Tokushima Bunri University, 8-53 Hamanocho, Takamatsu, Kagawa, 760-8542, Japan. tsubaki@kph.bunri-u.ac.jp., Obana T; Department of Pharmacy, Kindai University, 3-4-1 Kowakae, Higashi-Osaka, Osaka, 577-8502, Japan., Matsuo T; Laboratory of Pharmacotherapy, Faculty of Pharmaceutical Sciences at Kagawa Campus, Tokushima Bunri University, 8-53 Hamanocho, Takamatsu, Kagawa, 760-8542, Japan., Komori R; Laboratory of Pharmacotherapy, Faculty of Pharmaceutical Sciences at Kagawa Campus, Tokushima Bunri University, 8-53 Hamanocho, Takamatsu, Kagawa, 760-8542, Japan., Takeda T; Department of Pharmacy, Kindai University, 3-4-1 Kowakae, Higashi-Osaka, Osaka, 577-8502, Japan., Koumoto Y; Department of Pharmacy, Kindai University, 3-4-1 Kowakae, Higashi-Osaka, Osaka, 577-8502, Japan., Usami H; Department of Pharmacy, Kindai University, 3-4-1 Kowakae, Higashi-Osaka, Osaka, 577-8502, Japan., Nagai N; Department of Pharmacy, Kindai University, 3-4-1 Kowakae, Higashi-Osaka, Osaka, 577-8502, Japan., Nishida S; Department of Pharmacy, Kindai University, 3-4-1 Kowakae, Higashi-Osaka, Osaka, 577-8502, Japan. nishida@phar.kindai.ac.jp. |
| Source: | Medical oncology (Northwood, London, England) [Med Oncol] 2025 Jun 17; Vol. 42 (7), pp. 268. Date of Electronic Publication: 2025 Jun 17. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Springer Country of Publication: United States NLM ID: 9435512 Publication Model: Electronic Cited Medium: Internet ISSN: 1559-131X (Electronic) Linking ISSN: 13570560 NLM ISO Abbreviation: Med Oncol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
Be the first to leave a comment!
