View in EDS

Dominant-negative TP53 mutations potentiated by the HSF1-regulated proteostasis network.

Saved in:
Bibliographic Details
Title: Dominant-negative TP53 mutations potentiated by the HSF1-regulated proteostasis network.
Authors: Halim S; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Sebastian RM; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Liivak KE; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Patrick JE; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Hui T; Department of Chemistry, Tufts University, Medford, MA 02155, USA., Amici DR; Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Medical Scientist Training Program, Northwestern University Feinberg School of Medicine, Chicago, IL, USA., Giacomelli AO; Humber Polytechnic, Toronto, ON M9W 5L7, Canada., Rios P; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Butty VL; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; BioMicro Center, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Hahn WC; Dana-Farber Cancer Institute, Boston, MA 02215, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA., Sánchez-Rivera FJ; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Mendillo ML; Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA., Lin YS; Department of Chemistry, Tufts University, Medford, MA 02155, USA., Shoulders MD; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA. Electronic address: mshoulde@mit.edu.
Source: Molecular cell [Mol Cell] 2026 Jan 22; Vol. 86 (2), pp. 345-361.e6. Date of Electronic Publication: 2026 Jan 14.
Publication Type: Journal Article
Journal Info: Publisher: Cell Press Country of Publication: United States NLM ID: 9802571 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1097-4164 (Electronic) Linking ISSN: 10972765 NLM ISO Abbreviation: Mol Cell Subsets: MEDLINE
Database: MEDLINE Ultimate
FullText Text:
  Availability: 0
Header DbId: mdl
DbLabel: MEDLINE Ultimate
An: 41539306
AccessLevel: 2
PubType: Academic Journal
PubTypeId: academicJournal
PreciseRelevancyScore: 0
IllustrationInfo
Items – Name: Title
  Label: Title
  Group: Ti
  Data: Dominant-negative TP53 mutations potentiated by the HSF1-regulated proteostasis network.
– Name: Author
  Label: Authors
  Group: Au
  Data: <searchLink fieldCode="AU" term="%22Halim+S%22">Halim S</searchLink>; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Sebastian+RM%22">Sebastian RM</searchLink>; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Liivak+KE%22">Liivak KE</searchLink>; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Patrick+JE%22">Patrick JE</searchLink>; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Hui+T%22">Hui T</searchLink>; Department of Chemistry, Tufts University, Medford, MA 02155, USA.<br /><searchLink fieldCode="AU" term="%22Amici+DR%22">Amici DR</searchLink>; Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Medical Scientist Training Program, Northwestern University Feinberg School of Medicine, Chicago, IL, USA.<br /><searchLink fieldCode="AU" term="%22Giacomelli+AO%22">Giacomelli AO</searchLink>; Humber Polytechnic, Toronto, ON M9W 5L7, Canada.<br /><searchLink fieldCode="AU" term="%22Rios+P%22">Rios P</searchLink>; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Butty+VL%22">Butty VL</searchLink>; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; BioMicro Center, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Hahn+WC%22">Hahn WC</searchLink>; Dana-Farber Cancer Institute, Boston, MA 02215, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA.<br /><searchLink fieldCode="AU" term="%22Sánchez-Rivera+FJ%22">Sánchez-Rivera FJ</searchLink>; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA.<br /><searchLink fieldCode="AU" term="%22Mendillo+ML%22">Mendillo ML</searchLink>; Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA.<br /><searchLink fieldCode="AU" term="%22Lin+YS%22">Lin YS</searchLink>; Department of Chemistry, Tufts University, Medford, MA 02155, USA.<br /><searchLink fieldCode="AU" term="%22Shoulders+MD%22">Shoulders MD</searchLink>; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA. Electronic address: mshoulde@mit.edu.
– Name: TitleSource
  Label: Source
  Group: Src
  Data: <searchLink fieldCode="JN" term="%229802571%22">Molecular cell</searchLink> [Mol Cell] 2026 Jan 22; Vol. 86 (2), pp. 345-361.e6. <i>Date of Electronic Publication: </i>2026 Jan 14.
– Name: TypePub
  Label: Publication Type
  Group: TypPub
  Data: Journal Article
– Name: TitleSource
  Label: Journal Info
  Group: Src
  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Cell+Press%22">Cell Press </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>9802571 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1097-4164 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2210972765%22">10972765 </searchLink><i>NLM ISO Abbreviation: </i>Mol Cell <i>Subsets: </i>MEDLINE
PLink https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=41539306
RecordInfo BibRecord:
  BibEntity:
    Identifiers:
      – Type: doi
        Value: 10.1016/j.molcel.2025.12.013
    Languages:
      – Code: eng
        Text: English
    PhysicalDescription:
      Pagination:
        StartPage: 345
    Titles:
      – TitleFull: Dominant-negative TP53 mutations potentiated by the HSF1-regulated proteostasis network.
        Type: main
  BibRelationships:
    HasContributorRelationships:
      – PersonEntity:
          Name:
            NameFull: Halim S
      – PersonEntity:
          Name:
            NameFull: Sebastian RM
      – PersonEntity:
          Name:
            NameFull: Liivak KE
      – PersonEntity:
          Name:
            NameFull: Patrick JE
      – PersonEntity:
          Name:
            NameFull: Hui T
      – PersonEntity:
          Name:
            NameFull: Amici DR
      – PersonEntity:
          Name:
            NameFull: Giacomelli AO
      – PersonEntity:
          Name:
            NameFull: Rios P
      – PersonEntity:
          Name:
            NameFull: Butty VL
      – PersonEntity:
          Name:
            NameFull: Hahn WC
      – PersonEntity:
          Name:
            NameFull: Sánchez-Rivera FJ
      – PersonEntity:
          Name:
            NameFull: Mendillo ML
      – PersonEntity:
          Name:
            NameFull: Lin YS
      – PersonEntity:
          Name:
            NameFull: Shoulders MD
    IsPartOfRelationships:
      – BibEntity:
          Dates:
            – D: 22
              M: 01
              Text: 2026 Jan 22
              Type: published
              Y: 2026
          Identifiers:
            – Type: issn-electronic
              Value: 1097-4164
          Numbering:
            – Type: volume
              Value: 86
            – Type: issue
              Value: 2
          Titles:
            – TitleFull: Molecular cell
              Type: main
ResultId 1