Dominant-negative TP53 mutations potentiated by the HSF1-regulated proteostasis network.
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| Title: | Dominant-negative TP53 mutations potentiated by the HSF1-regulated proteostasis network. |
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| Authors: | Halim S; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Sebastian RM; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Liivak KE; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Patrick JE; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Hui T; Department of Chemistry, Tufts University, Medford, MA 02155, USA., Amici DR; Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Medical Scientist Training Program, Northwestern University Feinberg School of Medicine, Chicago, IL, USA., Giacomelli AO; Humber Polytechnic, Toronto, ON M9W 5L7, Canada., Rios P; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Butty VL; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; BioMicro Center, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Hahn WC; Dana-Farber Cancer Institute, Boston, MA 02215, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA., Sánchez-Rivera FJ; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Department of Biology, Massachusetts Institute of Technology, Cambridge, MA 02139, USA., Mendillo ML; Department of Biochemistry and Molecular Genetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Simpson Querrey Institute for Epigenetics, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA; Robert H. Lurie Comprehensive Cancer Center, Northwestern University Feinberg School of Medicine, Chicago, IL 60611, USA., Lin YS; Department of Chemistry, Tufts University, Medford, MA 02155, USA., Shoulders MD; Department of Chemistry, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, Cambridge, MA 02139, USA; Broad Institute of MIT and Harvard, Cambridge, MA 02142, USA. Electronic address: mshoulde@mit.edu. |
| Source: | Molecular cell [Mol Cell] 2026 Jan 22; Vol. 86 (2), pp. 345-361.e6. Date of Electronic Publication: 2026 Jan 14. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Cell Press Country of Publication: United States NLM ID: 9802571 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1097-4164 (Electronic) Linking ISSN: 10972765 NLM ISO Abbreviation: Mol Cell Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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