Multiple redundant mechanisms account for the majority of gene silencing downstream of DNA methylation.
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| Title: | Multiple redundant mechanisms account for the majority of gene silencing downstream of DNA methylation. |
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| Authors: | Wang S; Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Wu Z; Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA.; State Key Laboratory of Rice Biology and Breeding, Institute of Biotechnology, Zhejiang University, Hangzhou 310058, China.; Key Laboratory of Molecular Biology of Crop Pathogens and Insects, Zhejiang University, Hangzhou 310058, China.; Howard Hughes Medical Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA., Li Z; Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Movilli A; Department of Molecular Biology, Max Planck Institute for Biology Tübingen, Tübingen, Germany., He L; Department of Molecular Biology, Max Planck Institute for Biology Tübingen, Tübingen, Germany., Zhou Y; Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Lin EK; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Chuang R; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Thiri WW; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Convery S; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA., Feng S; Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Howard Hughes Medical Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA., Weigel D; Department of Molecular Biology, Max Planck Institute for Biology Tübingen, Tübingen, Germany., Jacobsen SE; Molecular Biology Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Department of Molecular, Cell and Developmental Biology, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Howard Hughes Medical Institute, University of California, Los Angeles, Los Angeles, CA 90095, USA.; Eli & Edythe Broad Center of Regenerative Medicine & Stem Cell Research, University of California at Los Angeles, Los Angeles, CA 90095, USA. |
| Source: | BioRxiv : the preprint server for biology [bioRxiv] 2026 Feb 10. Date of Electronic Publication: 2026 Feb 10. |
| Publication Type: | Journal Article; Preprint |
| Journal Info: | Country of Publication: United States NLM ID: 101680187 Publication Model: Electronic Cited Medium: Internet ISSN: 2692-8205 (Electronic) Linking ISSN: 26928205 NLM ISO Abbreviation: bioRxiv Subsets: PubMed not MEDLINE |
| Database: | MEDLINE Ultimate |
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