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The protein kinase DYRK1B is a p53 target gene and functions as a negative feedback regulator of the transcription factor RFX7.

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Title: The protein kinase DYRK1B is a p53 target gene and functions as a negative feedback regulator of the transcription factor RFX7.
Authors: Wilms G; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany., Schwandt K; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany., Düsterhöft S; Institute of Molecular Pharmacology, RWTH Aachen University, Aachen, Germany., Helmich P; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany., Wozniak J; Institute of Clinical Pharmacology, University Hospital of RWTH Aachen, Aachen, Germany., Kraft F; Medical Faculty, Center for Human Genetics and Genomic Medicine, RWTH University Aachen, Aachen, Germany., Kallabis S; Department of Systems Immunology & Proteomics, Institute of Innate Immunity, Medical Faculty, University of Bonn, Bonn, Germany., Meissner F; Department of Systems Immunology & Proteomics, Institute of Innate Immunity, Medical Faculty, University of Bonn, Bonn, Germany., Becker W; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany. wbecker@ukaachen.de.
Source: Cell death & disease [Cell Death Dis] 2026 Mar 26; Vol. 17 (1). Date of Electronic Publication: 2026 Mar 26.
Publication Type: Journal Article
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: The protein kinase DYRK1B is a p53 target gene and functions as a negative feedback regulator of the transcription factor RFX7.
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  Data: <searchLink fieldCode="AU" term="%22Wilms+G%22">Wilms G</searchLink>; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany.<br /><searchLink fieldCode="AU" term="%22Schwandt+K%22">Schwandt K</searchLink>; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany.<br /><searchLink fieldCode="AU" term="%22Düsterhöft+S%22">Düsterhöft S</searchLink>; Institute of Molecular Pharmacology, RWTH Aachen University, Aachen, Germany.<br /><searchLink fieldCode="AU" term="%22Helmich+P%22">Helmich P</searchLink>; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany.<br /><searchLink fieldCode="AU" term="%22Wozniak+J%22">Wozniak J</searchLink>; Institute of Clinical Pharmacology, University Hospital of RWTH Aachen, Aachen, Germany.<br /><searchLink fieldCode="AU" term="%22Kraft+F%22">Kraft F</searchLink>; Medical Faculty, Center for Human Genetics and Genomic Medicine, RWTH University Aachen, Aachen, Germany.<br /><searchLink fieldCode="AU" term="%22Kallabis+S%22">Kallabis S</searchLink>; Department of Systems Immunology & Proteomics, Institute of Innate Immunity, Medical Faculty, University of Bonn, Bonn, Germany.<br /><searchLink fieldCode="AU" term="%22Meissner+F%22">Meissner F</searchLink>; Department of Systems Immunology & Proteomics, Institute of Innate Immunity, Medical Faculty, University of Bonn, Bonn, Germany.<br /><searchLink fieldCode="AU" term="%22Becker+W%22">Becker W</searchLink>; Institute of Pharmacology and Toxicology, RWTH Aachen University, Aachen, Germany. wbecker@ukaachen.de.
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  Data: <searchLink fieldCode="JN" term="%22101524092%22">Cell death & disease</searchLink> [Cell Death Dis] 2026 Mar 26; Vol. 17 (1). <i>Date of Electronic Publication: </i>2026 Mar 26.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Pub%2E+Group%22">Nature Pub. Group </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101524092 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2041-4889 (Electronic) <i>NLM ISO Abbreviation: </i>Cell Death Dis <i>Subsets: </i>MEDLINE
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              Text: 2026 Mar 26
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