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NF-κB activation-induced anti-apoptosis renders HER2-positive cells drug resistant and accelerates tumor growth.

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Bibliographic Details
Title: NF-κB activation-induced anti-apoptosis renders HER2-positive cells drug resistant and accelerates tumor growth.
Authors: Bailey ST; Center for Functional Cancer Epigenetics, Dana-Farber Cancer Institute, Boston, MA 02115.; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA 02115., Miron PL; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115., Choi YJ; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115., Kochupurakkal B; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115., Maulik G; Center for Functional Cancer Epigenetics, Dana-Farber Cancer Institute, Boston, MA 02115., Rodig SJ; Department of Pathology, Brigham & Women's Hospital and Harvard Medical School, Boston, MA 02115., Tian R; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115., Foley KM; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115., Bowman T; Department of Pathology, Brigham & Women's Hospital and Harvard Medical School, Boston, MA 02115., Miron A; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115., Brown M; Center for Functional Cancer Epigenetics, Dana-Farber Cancer Institute, Boston, MA 02115.; Department of Medical Oncology, Dana-Farber Cancer Institute, Boston, MA 02115., Iglehart JD; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115.; Department of Surgery, Brigham & Women's Hospital and Harvard Medical School. Boston, MA 02115., Debajit KB; Department of Cancer Biology, Dana-Farber Cancer Institute & Harvard Medical School. Boston, MA 02115.; Department of Surgery, Brigham & Women's Hospital and Harvard Medical School. Boston, MA 02115.
Source: Molecular cancer research : MCR [Mol Cancer Res] 2014 Mar; Vol. 12 (3), pp. 408-420. Date of Electronic Publication: 2013 Dec 06.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: American Association for Cancer Research Country of Publication: United States NLM ID: 101150042 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1557-3125 (Electronic) Linking ISSN: 15417786 NLM ISO Abbreviation: Mol Cancer Res Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1557-3125
DOI:10.1158/1541-7786.MCR-13-0206-T