ABIN1 is a signal-induced autophagy receptor that attenuates NF-κB activation by recognizing linear ubiquitin chains.

Saved in:

Bibliographic Details
Title:	ABIN1 is a signal-induced autophagy receptor that attenuates NF-κB activation by recognizing linear ubiquitin chains.
Authors:	Shinkawa Y; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan., Imami K; Department of Molecular and Cellular BioAnalysis, Graduate School of Pharmaceutical Sciences, Kyoto University, Japan., Fuseya Y; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan., Sasaki K; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan., Ohmura K; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan., Ishihama Y; Department of Molecular and Cellular BioAnalysis, Graduate School of Pharmaceutical Sciences, Kyoto University, Japan., Morinobu A; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan., Iwai K; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.
Source:	FEBS letters [FEBS Lett] 2022 May; Vol. 596 (9), pp. 1147-1164. Date of Electronic Publication: 2022 Mar 04.
Publication Type:	Journal Article; Research Support, Non-U.S. Gov't
Journal Info:	Publisher: John Wiley & Sons Ltd Country of Publication: England NLM ID: 0155157 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1873-3468 (Electronic) Linking ISSN: 00145793 NLM ISO Abbreviation: FEBS Lett Subsets: MEDLINE
Database:	MEDLINE Ultimate

Description
ISSN:	1873-3468
DOI:	10.1002/1873-3468.14323