ABIN1 is a signal-induced autophagy receptor that attenuates NF-κB activation by recognizing linear ubiquitin chains.

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Title: ABIN1 is a signal-induced autophagy receptor that attenuates NF-κB activation by recognizing linear ubiquitin chains.
Authors: Shinkawa Y; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan., Imami K; Department of Molecular and Cellular BioAnalysis, Graduate School of Pharmaceutical Sciences, Kyoto University, Japan., Fuseya Y; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan., Sasaki K; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan., Ohmura K; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan., Ishihama Y; Department of Molecular and Cellular BioAnalysis, Graduate School of Pharmaceutical Sciences, Kyoto University, Japan., Morinobu A; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan., Iwai K; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.
Source: FEBS letters [FEBS Lett] 2022 May; Vol. 596 (9), pp. 1147-1164. Date of Electronic Publication: 2022 Mar 04.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: John Wiley & Sons Ltd Country of Publication: England NLM ID: 0155157 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1873-3468 (Electronic) Linking ISSN: 00145793 NLM ISO Abbreviation: FEBS Lett Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: ABIN1 is a signal-induced autophagy receptor that attenuates NF-κB activation by recognizing linear ubiquitin chains.
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  Data: <searchLink fieldCode="AU" term="%22Shinkawa+Y%22">Shinkawa Y</searchLink>; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Imami+K%22">Imami K</searchLink>; Department of Molecular and Cellular BioAnalysis, Graduate School of Pharmaceutical Sciences, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Fuseya+Y%22">Fuseya Y</searchLink>; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Sasaki+K%22">Sasaki K</searchLink>; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Ohmura+K%22">Ohmura K</searchLink>; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Ishihama+Y%22">Ishihama Y</searchLink>; Department of Molecular and Cellular BioAnalysis, Graduate School of Pharmaceutical Sciences, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Morinobu+A%22">Morinobu A</searchLink>; Department of Rheumatology and Clinical Immunology, Graduate School of Medicine, Kyoto University, Japan.<br /><searchLink fieldCode="AU" term="%22Iwai+K%22">Iwai K</searchLink>; Department of Molecular and Cellular Physiology, Graduate School of Medicine, Kyoto University, Japan.
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  Data: <searchLink fieldCode="JN" term="%220155157%22">FEBS letters</searchLink> [FEBS Lett] 2022 May; Vol. 596 (9), pp. 1147-1164. <i>Date of Electronic Publication: </i>2022 Mar 04.
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  Data: Journal Article; Research Support, Non-U.S. Gov't
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22John+Wiley+%26+Sons+Ltd%22">John Wiley & Sons Ltd </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>0155157 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1873-3468 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200145793%22">00145793 </searchLink><i>NLM ISO Abbreviation: </i>FEBS Lett <i>Subsets: </i>MEDLINE
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        Value: 10.1002/1873-3468.14323
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        Text: English
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      – TitleFull: ABIN1 is a signal-induced autophagy receptor that attenuates NF-κB activation by recognizing linear ubiquitin chains.
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              Text: 2022 May
              Type: published
              Y: 2022
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