Disruption of mitochondria-sarcoplasmic reticulum microdomain connectomics contributes to sinus node dysfunction in heart failure.

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Bibliographic Details
Title: Disruption of mitochondria-sarcoplasmic reticulum microdomain connectomics contributes to sinus node dysfunction in heart failure.
Authors: Ren L; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305., Gopireddy RR; Department of Pharmacology, University of California, Davis, CA 95616., Perkins G; National Center for Microscopy and Imaging Research, University of California San Diego, La Jolla, CA 92093., Zhang H; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305., Timofeyev V; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Lyu Y; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Diloretto DA; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Trinh P; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Sirish P; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Overton JL; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Xu W; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Grainger N; Department of Physiology and Membrane Biology, University of California, Davis, CA 95616., Xiang YK; Department of Pharmacology, University of California, Davis, CA 95616., Dedkova EN; Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, CA 95616., Zhang XD; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Yamoah EN; Department of Physiology and Cell Biology, University of Nevada, Reno, NV 89557., Navedo MF; Department of Pharmacology, University of California, Davis, CA 95616., Thai PN; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Department of Physiology and Cell Biology, University of Nevada, Reno, NV 89557., Chiamvimonvat N; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Department of Pharmacology, University of California, Davis, CA 95616.; Department of Veterans Affairs, Northern California Health Care System, Mather, CA 95655.
Source: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2022 Sep 06; Vol. 119 (36), pp. e2206708119. Date of Electronic Publication: 2022 Aug 31.
Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1091-6490 (Electronic) Linking ISSN: 00278424 NLM ISO Abbreviation: Proc Natl Acad Sci U S A Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1091-6490
DOI:10.1073/pnas.2206708119