Disruption of mitochondria-sarcoplasmic reticulum microdomain connectomics contributes to sinus node dysfunction in heart failure.
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| Title: | Disruption of mitochondria-sarcoplasmic reticulum microdomain connectomics contributes to sinus node dysfunction in heart failure. |
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| Authors: | Ren L; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305., Gopireddy RR; Department of Pharmacology, University of California, Davis, CA 95616., Perkins G; National Center for Microscopy and Imaging Research, University of California San Diego, La Jolla, CA 92093., Zhang H; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305., Timofeyev V; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Lyu Y; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Diloretto DA; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Trinh P; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Sirish P; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Overton JL; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Xu W; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Grainger N; Department of Physiology and Membrane Biology, University of California, Davis, CA 95616., Xiang YK; Department of Pharmacology, University of California, Davis, CA 95616., Dedkova EN; Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, CA 95616., Zhang XD; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616., Yamoah EN; Department of Physiology and Cell Biology, University of Nevada, Reno, NV 89557., Navedo MF; Department of Pharmacology, University of California, Davis, CA 95616., Thai PN; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Department of Physiology and Cell Biology, University of Nevada, Reno, NV 89557., Chiamvimonvat N; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Department of Pharmacology, University of California, Davis, CA 95616.; Department of Veterans Affairs, Northern California Health Care System, Mather, CA 95655. |
| Source: | Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2022 Sep 06; Vol. 119 (36), pp. e2206708119. Date of Electronic Publication: 2022 Aug 31. |
| Publication Type: | Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: National Academy of Sciences Country of Publication: United States NLM ID: 7505876 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1091-6490 (Electronic) Linking ISSN: 00278424 NLM ISO Abbreviation: Proc Natl Acad Sci U S A Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
| FullText | Text: Availability: 0 |
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| Header | DbId: mdl DbLabel: MEDLINE Ultimate An: 36044551 AccessLevel: 2 PubType: Academic Journal PubTypeId: academicJournal PreciseRelevancyScore: 0 |
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| Items | – Name: Title Label: Title Group: Ti Data: Disruption of mitochondria-sarcoplasmic reticulum microdomain connectomics contributes to sinus node dysfunction in heart failure. – Name: Author Label: Authors Group: Au Data: <searchLink fieldCode="AU" term="%22Ren+L%22">Ren L</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Gopireddy+RR%22">Gopireddy RR</searchLink>; Department of Pharmacology, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Perkins+G%22">Perkins G</searchLink>; National Center for Microscopy and Imaging Research, University of California San Diego, La Jolla, CA 92093.<br /><searchLink fieldCode="AU" term="%22Zhang+H%22">Zhang H</searchLink>; Stanford Cardiovascular Institute, Stanford University School of Medicine, Stanford, CA 94305.<br /><searchLink fieldCode="AU" term="%22Timofeyev+V%22">Timofeyev V</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Lyu+Y%22">Lyu Y</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Diloretto+DA%22">Diloretto DA</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Trinh+P%22">Trinh P</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Sirish+P%22">Sirish P</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Overton+JL%22">Overton JL</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Xu+W%22">Xu W</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Grainger+N%22">Grainger N</searchLink>; Department of Physiology and Membrane Biology, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Xiang+YK%22">Xiang YK</searchLink>; Department of Pharmacology, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Dedkova+EN%22">Dedkova EN</searchLink>; Department of Molecular Biosciences, School of Veterinary Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Zhang+XD%22">Zhang XD</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Yamoah+EN%22">Yamoah EN</searchLink>; Department of Physiology and Cell Biology, University of Nevada, Reno, NV 89557.<br /><searchLink fieldCode="AU" term="%22Navedo+MF%22">Navedo MF</searchLink>; Department of Pharmacology, University of California, Davis, CA 95616.<br /><searchLink fieldCode="AU" term="%22Thai+PN%22">Thai PN</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Department of Physiology and Cell Biology, University of Nevada, Reno, NV 89557.<br /><searchLink fieldCode="AU" term="%22Chiamvimonvat+N%22">Chiamvimonvat N</searchLink>; Division of Cardiovascular Medicine, Department of Internal Medicine, University of California, Davis, CA 95616.; Department of Pharmacology, University of California, Davis, CA 95616.; Department of Veterans Affairs, Northern California Health Care System, Mather, CA 95655. – Name: TitleSource Label: Source Group: Src Data: <searchLink fieldCode="JN" term="%227505876%22">Proceedings of the National Academy of Sciences of the United States of America</searchLink> [Proc Natl Acad Sci U S A] 2022 Sep 06; Vol. 119 (36), pp. e2206708119. <i>Date of Electronic Publication: </i>2022 Aug 31. – Name: TypePub Label: Publication Type Group: TypPub Data: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't – Name: TitleSource Label: Journal Info Group: Src Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22National+Academy+of+Sciences%22">National Academy of Sciences </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>7505876 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1091-6490 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200278424%22">00278424 </searchLink><i>NLM ISO Abbreviation: </i>Proc Natl Acad Sci U S A <i>Subsets: </i>MEDLINE |
| PLink | https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&db=mdl&AN=36044551 |
| RecordInfo | BibRecord: BibEntity: Identifiers: – Type: doi Value: 10.1073/pnas.2206708119 Languages: – Code: eng Text: English PhysicalDescription: Pagination: StartPage: e2206708119 Titles: – TitleFull: Disruption of mitochondria-sarcoplasmic reticulum microdomain connectomics contributes to sinus node dysfunction in heart failure. Type: main BibRelationships: HasContributorRelationships: – PersonEntity: Name: NameFull: Ren L – PersonEntity: Name: NameFull: Gopireddy RR – PersonEntity: Name: NameFull: Perkins G – PersonEntity: Name: NameFull: Zhang H – PersonEntity: Name: NameFull: Timofeyev V – PersonEntity: Name: NameFull: Lyu Y – PersonEntity: Name: NameFull: Diloretto DA – PersonEntity: Name: NameFull: Trinh P – PersonEntity: Name: NameFull: Sirish P – PersonEntity: Name: NameFull: Overton JL – PersonEntity: Name: NameFull: Xu W – PersonEntity: Name: NameFull: Grainger N – PersonEntity: Name: NameFull: Xiang YK – PersonEntity: Name: NameFull: Dedkova EN – PersonEntity: Name: NameFull: Zhang XD – PersonEntity: Name: NameFull: Yamoah EN – PersonEntity: Name: NameFull: Navedo MF – PersonEntity: Name: NameFull: Thai PN – PersonEntity: Name: NameFull: Chiamvimonvat N IsPartOfRelationships: – BibEntity: Dates: – D: 06 M: 09 Text: 2022 Sep 06 Type: published Y: 2022 Identifiers: – Type: issn-electronic Value: 1091-6490 Numbering: – Type: volume Value: 119 – Type: issue Value: 36 Titles: – TitleFull: Proceedings of the National Academy of Sciences of the United States of America Type: main |
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