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Autophagy inhibition impairs leukemia stem cell function in FLT3-ITD AML but has antagonistic interactions with tyrosine kinase inhibition.

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Title: Autophagy inhibition impairs leukemia stem cell function in FLT3-ITD AML but has antagonistic interactions with tyrosine kinase inhibition.
Authors: Qiu S; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.; State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood diseases, Haihe Laboratory of Cell Ecosystem, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Science and Peking Union Medical College, Tianjin, China., Kumar H; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Yan C; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Li H; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Paterson AJ; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Anderson NR; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., He J; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Yang J; Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA., Xie M; Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA., Crossman DK; Genomics Core Facility, University of Alabama at Birmingham, Birmingham, AL, USA., Lu R; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Welner RS; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA., Bhatia R; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA. rbhatia@uabmc.edu.
Source: Leukemia [Leukemia] 2022 Nov; Vol. 36 (11), pp. 2621-2633. Date of Electronic Publication: 2022 Oct 11.
Publication Type: Journal Article; Research Support, N.I.H., Extramural
Journal Info: Publisher: Nature Publishing Group, Specialist Journals Country of Publication: England NLM ID: 8704895 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1476-5551 (Electronic) Linking ISSN: 08876924 NLM ISO Abbreviation: Leukemia Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Autophagy inhibition impairs leukemia stem cell function in FLT3-ITD AML but has antagonistic interactions with tyrosine kinase inhibition.
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  Data: <searchLink fieldCode="AU" term="%22Qiu+S%22">Qiu S</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.; State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood diseases, Haihe Laboratory of Cell Ecosystem, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Science and Peking Union Medical College, Tianjin, China.<br /><searchLink fieldCode="AU" term="%22Kumar+H%22">Kumar H</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Yan+C%22">Yan C</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Li+H%22">Li H</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Paterson+AJ%22">Paterson AJ</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Anderson+NR%22">Anderson NR</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22He+J%22">He J</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Yang+J%22">Yang J</searchLink>; Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Xie+M%22">Xie M</searchLink>; Division of Cardiovascular Disease, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Crossman+DK%22">Crossman DK</searchLink>; Genomics Core Facility, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Lu+R%22">Lu R</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Welner+RS%22">Welner RS</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA.<br /><searchLink fieldCode="AU" term="%22Bhatia+R%22">Bhatia R</searchLink>; Division of Hematology and Oncology, University of Alabama at Birmingham, Birmingham, AL, USA. rbhatia@uabmc.edu.
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  Data: <searchLink fieldCode="JN" term="%228704895%22">Leukemia</searchLink> [Leukemia] 2022 Nov; Vol. 36 (11), pp. 2621-2633. <i>Date of Electronic Publication: </i>2022 Oct 11.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Publishing+Group%2C+Specialist+Journals%22">Nature Publishing Group, Specialist Journals </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>8704895 <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1476-5551 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2208876924%22">08876924 </searchLink><i>NLM ISO Abbreviation: </i>Leukemia <i>Subsets: </i>MEDLINE
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