Microglia-mediated T cell infiltration drives neurodegeneration in tauopathy.

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Bibliographic Details
Title: Microglia-mediated T cell infiltration drives neurodegeneration in tauopathy.
Authors: Chen X; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Firulyova M; Almazov National Medical Research Centre, St Petersburg, Russia., Manis M; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Herz J; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA.; Center for Brain Immunology and Glia (BIG), Washington University School of Medicine, St Louis, MO, USA., Smirnov I; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA.; Center for Brain Immunology and Glia (BIG), Washington University School of Medicine, St Louis, MO, USA., Aladyeva E; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA., Wang C; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Bao X; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Finn MB; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Hu H; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Shchukina I; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA., Kim MW; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA.; Center for Brain Immunology and Glia (BIG), Washington University School of Medicine, St Louis, MO, USA., Yuede CM; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Kipnis J; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA.; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA.; Center for Brain Immunology and Glia (BIG), Washington University School of Medicine, St Louis, MO, USA., Artyomov MN; Department of Pathology and Immunology, Washington University School of Medicine, St Louis, MO, USA., Ulrich JD; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA., Holtzman DM; Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University School of Medicine, St Louis, MO, USA. holtzman@wustl.edu.; Center for Brain Immunology and Glia (BIG), Washington University School of Medicine, St Louis, MO, USA. holtzman@wustl.edu.
Source: Nature [Nature] 2023 Mar; Vol. 615 (7953), pp. 668-677. Date of Electronic Publication: 2023 Mar 08.
Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Publishing Group Country of Publication: England NLM ID: 0410462 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1476-4687 (Electronic) Linking ISSN: 00280836 NLM ISO Abbreviation: Nature Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1476-4687
DOI:10.1038/s41586-023-05788-0