Stress deficits in reward behaviour are associated with and replicated by dysregulated amygdala-nucleus accumbens pathway function in mice.

Saved in:
Bibliographic Details
Title: Stress deficits in reward behaviour are associated with and replicated by dysregulated amygdala-nucleus accumbens pathway function in mice.
Authors: Madur L; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland.; Zurich Neuroscience Center, University of Zurich and ETH Zurich, Zurich, Switzerland., Ineichen C; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland., Bergamini G; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland., Greter A; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland., Poggi G; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland., Cuomo-Haymour N; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland.; Zurich Neuroscience Center, University of Zurich and ETH Zurich, Zurich, Switzerland., Sigrist H; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland., Sych Y; Institute of Cellular and Integrative Neuroscience, University of Strasbourg, Strasbourg, France., Paterna JC; Viral Vector Facility, University of Zurich and ETH Zurich, Zurich, Switzerland., Bornemann KD; CNS Diseases Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany., Viollet C; Global Computational Biology and Digital Sciences, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany., Fernandez-Albert F; Global Computational Biology and Digital Sciences, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany., Alanis-Lobato G; Global Computational Biology and Digital Sciences, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany., Hengerer B; CNS Diseases Research, Boehringer Ingelheim Pharma GmbH & Co. KG, Biberach, Germany., Pryce CR; Preclinical Laboratory, Department of Psychiatry, Psychotherapy and Psychosomatics, Psychiatric University Hospital Zürich (PUK) and University of Zurich (UZH), Zurich, Switzerland. christopher.pryce@bli.uzh.ch.; Zurich Neuroscience Center, University of Zurich and ETH Zurich, Zurich, Switzerland. christopher.pryce@bli.uzh.ch.
Source: Communications biology [Commun Biol] 2023 Apr 15; Vol. 6 (1), pp. 422. Date of Electronic Publication: 2023 Apr 15.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Publishing Group UK Country of Publication: England NLM ID: 101719179 Publication Model: Electronic Cited Medium: Internet ISSN: 2399-3642 (Electronic) Linking ISSN: 23993642 NLM ISO Abbreviation: Commun Biol Subsets: MEDLINE
Database: MEDLINE Ultimate
Full text is not displayed to guests.
Description
ISSN:2399-3642
DOI:10.1038/s42003-023-04811-4