Glucolipotoxic Stress-Induced Mig6 Desensitizes EGFR Signaling and Promotes Pancreatic Beta Cell Death.

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Title: Glucolipotoxic Stress-Induced Mig6 Desensitizes EGFR Signaling and Promotes Pancreatic Beta Cell Death.
Authors: Chen YC; Department of Pediatrics and Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Cellular & Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA., Lutkewitte AJ; Department of Pediatrics and Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Cellular & Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA., Basavarajappa HD; Department of Molecular and Cellular Endocrinology, Beckman Research Institute of the City of Hope, Duarte, CA 91010, USA., Fueger PT; Department of Pediatrics and Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Cellular & Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Molecular and Cellular Endocrinology, Beckman Research Institute of the City of Hope, Duarte, CA 91010, USA.
Source: Metabolites [Metabolites] 2023 May 04; Vol. 13 (5). Date of Electronic Publication: 2023 May 04.
Publication Type: Journal Article
Journal Info: Publisher: MDPI Country of Publication: Switzerland NLM ID: 101578790 Publication Model: Electronic Cited Medium: Print ISSN: 2218-1989 (Print) Linking ISSN: 22181989 NLM ISO Abbreviation: Metabolites Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
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  Data: Glucolipotoxic Stress-Induced Mig6 Desensitizes EGFR Signaling and Promotes Pancreatic Beta Cell Death.
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  Data: <searchLink fieldCode="AU" term="%22Chen+YC%22">Chen YC</searchLink>; Department of Pediatrics and Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Cellular & Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA.<br /><searchLink fieldCode="AU" term="%22Lutkewitte+AJ%22">Lutkewitte AJ</searchLink>; Department of Pediatrics and Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Cellular & Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA.<br /><searchLink fieldCode="AU" term="%22Basavarajappa+HD%22">Basavarajappa HD</searchLink>; Department of Molecular and Cellular Endocrinology, Beckman Research Institute of the City of Hope, Duarte, CA 91010, USA.<br /><searchLink fieldCode="AU" term="%22Fueger+PT%22">Fueger PT</searchLink>; Department of Pediatrics and Herman B. Wells Center for Pediatric Research, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Cellular & Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA.; Department of Molecular and Cellular Endocrinology, Beckman Research Institute of the City of Hope, Duarte, CA 91010, USA.
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  Data: <searchLink fieldCode="JN" term="%22101578790%22">Metabolites</searchLink> [Metabolites] 2023 May 04; Vol. 13 (5). <i>Date of Electronic Publication: </i>2023 May 04.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22MDPI%22">MDPI </searchLink><i>Country of Publication: </i>Switzerland <i>NLM ID: </i>101578790 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Print <i>ISSN: </i>2218-1989 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2222181989%22">22181989 </searchLink><i>NLM ISO Abbreviation: </i>Metabolites <i>Subsets: </i>PubMed not MEDLINE
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        Value: 10.3390/metabo13050627
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        Text: English
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      – TitleFull: Glucolipotoxic Stress-Induced Mig6 Desensitizes EGFR Signaling and Promotes Pancreatic Beta Cell Death.
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            NameFull: Chen YC
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            NameFull: Lutkewitte AJ
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            – D: 04
              M: 05
              Text: 2023 May 04
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              Y: 2023
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