Dominant dystrophic epidermolysis bullosa is associated with glycolytically active GATA3+ T helper 2 cells which may contribute to pruritus in lesional skin.
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| Title: | Dominant dystrophic epidermolysis bullosa is associated with glycolytically active GATA3+ T helper 2 cells which may contribute to pruritus in lesional skin. |
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| Authors: | Aala WJF; Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan., Hou PC; Department of Dermatology, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan., Hong YK; Department of Dermatology, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; International Center for Wound Repair and Regeneration, National Cheng Kung University, Tainan, Taiwan., Lin YC; Department of Dermatology, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; International Center for Wound Repair and Regeneration, National Cheng Kung University, Tainan, Taiwan., Lee YR; School of Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan., Tu WT; Department of Dermatology, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan., Papanikolaou M; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK., Benzian-Olsson N; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK., Onoufriadis A; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK.; Laboratory of Medical Biology and Genetics, School of Medicine, Aristotle University of Thessaloniki, Thessaloniki, Greece., I Chen Harn H; Department of Pathology, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA., Hwang DY; National Institute of Cancer Research, National Health Research Institutes, Tainan, Taiwan., Cheng SM; National Institute of Cancer Research, National Health Research Institutes, Tainan, Taiwan., Lu K; Department of Dermatology, Feinberg School of Medicine, Northwestern University, Chicago, IL, USA., Chen PC; Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan., McGrath JA; School of Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; St John's Institute of Dermatology, School of Basic and Medical Biosciences, King's College London, London, UK., Hsu CK; Institute of Clinical Medicine, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; Department of Dermatology, National Cheng Kung University Hospital, College of Medicine, National Cheng Kung University, Tainan, Taiwan.; International Center for Wound Repair and Regeneration, National Cheng Kung University, Tainan, Taiwan. |
| Source: | The British journal of dermatology [Br J Dermatol] 2024 Jul 16; Vol. 191 (2), pp. 252-260. |
| Publication Type: | Journal Article; Research Support, Non-U.S. Gov't |
| Journal Info: | Publisher: Oxford University Press Country of Publication: England NLM ID: 0004041 Publication Model: Print Cited Medium: Internet ISSN: 1365-2133 (Electronic) Linking ISSN: 00070963 NLM ISO Abbreviation: Br J Dermatol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 1365-2133 |
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| DOI: | 10.1093/bjd/ljae110 |
