Synaptic cell adhesion molecules contribute to the pathogenesis and progression of fragile X syndrome.

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Title: Synaptic cell adhesion molecules contribute to the pathogenesis and progression of fragile X syndrome.
Authors: Bai SY; Geriatric Hospital Affiliated to Wuhan University of Science and Technology, Wuhan, China.; Hubei Provincial Clinical Research Center for Alzheimer's Disease, Wuhan University of Science and Technology, Wuhan, China., Zeng DY; Geriatric Hospital Affiliated to Wuhan University of Science and Technology, Wuhan, China.; Hubei Provincial Clinical Research Center for Alzheimer's Disease, Wuhan University of Science and Technology, Wuhan, China., Ouyang M; Geriatric Hospital Affiliated to Wuhan University of Science and Technology, Wuhan, China.; Hubei Provincial Clinical Research Center for Alzheimer's Disease, Wuhan University of Science and Technology, Wuhan, China., Zeng Y; Geriatric Hospital Affiliated to Wuhan University of Science and Technology, Wuhan, China.; Hubei Provincial Clinical Research Center for Alzheimer's Disease, Wuhan University of Science and Technology, Wuhan, China., Tan W; Geriatric Hospital Affiliated to Wuhan University of Science and Technology, Wuhan, China.; Hubei Provincial Clinical Research Center for Alzheimer's Disease, Wuhan University of Science and Technology, Wuhan, China., Xu L; Geriatric Hospital Affiliated to Wuhan University of Science and Technology, Wuhan, China.; Hubei Provincial Clinical Research Center for Alzheimer's Disease, Wuhan University of Science and Technology, Wuhan, China.
Source: Frontiers in cellular neuroscience [Front Cell Neurosci] 2024 Jul 03; Vol. 18, pp. 1393536. Date of Electronic Publication: 2024 Jul 03 (Print Publication: 2024).
Publication Type: Journal Article; Review
Journal Info: Publisher: Frontiers Research Foundation Country of Publication: Switzerland NLM ID: 101477935 Publication Model: eCollection Cited Medium: Print ISSN: 1662-5102 (Print) Linking ISSN: 16625102 NLM ISO Abbreviation: Front Cell Neurosci Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1662-5102
DOI:10.3389/fncel.2024.1393536