Mechanics-activated fibroblasts promote pulmonary group 2 innate lymphoid cell plasticity propelling silicosis progression.

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Title: Mechanics-activated fibroblasts promote pulmonary group 2 innate lymphoid cell plasticity propelling silicosis progression.
Authors: He Y; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China.; Department of Microbiology, Immunology and Molecular Genetics, University of Texas Health Science Center San Antonio, San Antonio, TX, USA., Yang F; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China., Yang L; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China., Yuan H; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China., You Y; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China., Chen Y; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China., Wu X; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China., Min H; Department of Immunology, College of Basic Medical Sciences, China Medical University, Shenyang, Liaoning, PR China., Chen J; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China. jchen@cmu.edu.cn.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China. jchen@cmu.edu.cn., Li C; Key Laboratory of Environmental Stress and Chronic Disease Control & Prevention (China Medical University), Ministry of Education, Shenyang, Liaoning, PR China. lichao@cmu.edu.cn.; Department of Occupational and Environmental Health, School of Public Health, China Medical University, Shenyang, Liaoning, PR China. lichao@cmu.edu.cn.
Source: Nature communications [Nat Commun] 2024 Nov 12; Vol. 15 (1), pp. 9770. Date of Electronic Publication: 2024 Nov 12.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE
Database: MEDLINE Ultimate
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ISSN:2041-1723
DOI:10.1038/s41467-024-54174-5