Perivascular glial reactivity is a feature of phosphorylated tau lesions in chronic traumatic encephalopathy.

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Title: Perivascular glial reactivity is a feature of phosphorylated tau lesions in chronic traumatic encephalopathy.
Authors: Osterman C; Department of Anatomy and Medical Imaging and Centre for Brain Research, Faculty of Medical and Health Science, University of Auckland, 85 Park Road, Grafton, 1023, Auckland, New Zealand., Hamlin D; Department of Anatomy and Medical Imaging and Centre for Brain Research, Faculty of Medical and Health Science, University of Auckland, 85 Park Road, Grafton, 1023, Auckland, New Zealand., Suter CM; Department of Neuropathology, Royal Prince Alfred Hospital, 94 Mallet St, Camperdown, NSW, 2050, Australia.; School of Medical Sciences, Faculty of Medicine and Health, University of Sydney, Camperdown, NSW, 2006, Australia., Affleck AJ; Department of Neuropathology, Royal Prince Alfred Hospital, 94 Mallet St, Camperdown, NSW, 2050, Australia.; School of Medical Sciences, Faculty of Medicine and Health, University of Sydney, Camperdown, NSW, 2006, Australia., Gloss BS; Westmead Research Hub, Westmead Institute for Medical Research, Westmead, NSW, Australia., Turner CP; Department of Anatomy and Medical Imaging and Centre for Brain Research, Faculty of Medical and Health Science, University of Auckland, 85 Park Road, Grafton, 1023, Auckland, New Zealand.; Department of Anatomical Pathology, Pathology and Laboratory Medicine, Auckland City Hospital, 2 Park Road, Grafton, 1023, Auckland, New Zealand., Faull RLM; Department of Anatomy and Medical Imaging and Centre for Brain Research, Faculty of Medical and Health Science, University of Auckland, 85 Park Road, Grafton, 1023, Auckland, New Zealand., Stein TD; Department of Pathology and Laboratory Medicine, VA Boston Healthcare System, Boston, MA, USA.; Department of Pathology, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Alzheimer's Disease and CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Bedford Veterans Affairs Medical Center, Bedford, MA, USA., McKee A; Department of Pathology and Laboratory Medicine, VA Boston Healthcare System, Boston, MA, USA.; Department of Pathology, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Alzheimer's Disease and CTE Center, Boston University Chobanian & Avedisian School of Medicine, Boston, MA, USA.; Bedford Veterans Affairs Medical Center, Bedford, MA, USA., Buckland ME; Department of Neuropathology, Royal Prince Alfred Hospital, 94 Mallet St, Camperdown, NSW, 2050, Australia.; School of Medical Sciences, Faculty of Medicine and Health, University of Sydney, Camperdown, NSW, 2006, Australia., Curtis MA; Department of Anatomy and Medical Imaging and Centre for Brain Research, Faculty of Medical and Health Science, University of Auckland, 85 Park Road, Grafton, 1023, Auckland, New Zealand., Murray HC; Department of Anatomy and Medical Imaging and Centre for Brain Research, Faculty of Medical and Health Science, University of Auckland, 85 Park Road, Grafton, 1023, Auckland, New Zealand. h.murray@auckland.ac.nz.
Source: Acta neuropathologica [Acta Neuropathol] 2025 Feb 08; Vol. 149 (1), pp. 16. Date of Electronic Publication: 2025 Feb 08.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Springer Verlag Country of Publication: Germany NLM ID: 0412041 Publication Model: Electronic Cited Medium: Internet ISSN: 1432-0533 (Electronic) Linking ISSN: 00016322 NLM ISO Abbreviation: Acta Neuropathol Subsets: MEDLINE
Database: MEDLINE Ultimate
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Description
ISSN:1432-0533
DOI:10.1007/s00401-025-02854-x