PHD3-VHL axis controls HIV-2 infection through oxygen-dependent hydroxylation and degradation of Vpx.

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Title: PHD3-VHL axis controls HIV-2 infection through oxygen-dependent hydroxylation and degradation of Vpx.
Authors: Miyakawa K; AIDS Research Center, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan.; Influenza Research Center, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan.; Department of Microbiology, Yokohama City University School of Medicine, Yokohama, Japan., Tanaka K; Department of Microbiology, Yokohama City University School of Medicine, Yokohama, Japan., Ino Y; Advanced Medical Research Center, Yokohama City University, Yokohama, Japan., Kimura Y; Advanced Medical Research Center, Yokohama City University, Yokohama, Japan., Kameya T; Department of Bioinformatics and Integrative Omics, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan.; Life Science Laboratory, Technology and Development Division, Kanto Chemical Co., Inc., Isehara, Japan., Mizukoshi F; Department of Bioinformatics and Integrative Omics, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan., Nishi M; Department of Microbiology, Yokohama City University School of Medicine, Yokohama, Japan.; Department of Bioinformatics and Integrative Omics, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan., Yokoyama M; Department of Bioinformatics and Integrative Omics, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan., Nakabayashi J; Institute of Liberal Arts, Institute of Science Tokyo, Ichikawa, Japan., Nomaguchi M; Department of Microbiology, Tokushima University Graduate School of Biomedical Sciences, Tokushima, Japan., Sato H; Department of Bioinformatics and Integrative Omics, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan., Kimura H; Department of Health Science, Graduate School of Health Sciences, Gunma Paz University, Takasaki, Japan., Akari H; Center for the Evolutionary Origins of Human Behavior, Kyoto University, Inuyama, Japan., Miura T; Institute for Life and Medical Sciences, Kyoto University, Kyoto, Japan., Takaoka A; Division of Signaling in Cancer and Immunology, Institute for Genetic Medicine, Hokkaido University, Sapporo, Japan., Hasegawa H; Influenza Research Center, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan.; Institute for Vaccine Research and Development, Hokkaido University, Sapporo, Japan., Matano T; AIDS Research Center, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan.; Institute of Medical Science, University of Tokyo, Tokyo, Japan., Minamishima YA; Department of Biochemistry, Gunma University Graduate School of Medicine, Maebashi, Japan., Ryo A; Department of Microbiology, Yokohama City University School of Medicine, Yokohama, Japan.; Department of Bioinformatics and Integrative Omics, National Institute of Infectious Diseases, Japan Institute for Health Security, Tokyo, Japan.
Source: PLoS pathogens [PLoS Pathog] 2025 Jun 16; Vol. 21 (6), pp. e1013241. Date of Electronic Publication: 2025 Jun 16 (Print Publication: 2025).
Publication Type: Journal Article
Journal Info: Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE
Database: MEDLINE Ultimate
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ISSN:1553-7374
DOI:10.1371/journal.ppat.1013241