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Cancer-associated fibroblasts promote drug resistance in ALK-driven lung adenocarcinoma cells by upregulating lipid biosynthesis.

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Title: Cancer-associated fibroblasts promote drug resistance in ALK-driven lung adenocarcinoma cells by upregulating lipid biosynthesis.
Authors: Daum AK; Division of Cancer Genome Research, German Cancer Research Center (DKFZ), German Cancer Consortium (DKTK), National Center for Tumor Diseases (NCT), Im Neuenheimer Feld 280, 69120, Heidelberg, Germany.; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Medical Faculty, Heidelberg University, Heidelberg, Germany., Schlicker L; Division of Tumor Metabolism and Microenvironment, German Cancer Research Center (DKFZ), Heidelberg, Germany.; Proteomics Core Facility, German Cancer Research Center (DKFZ), Heidelberg, Germany., Schneider MA; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Translational Research Unit, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany., Muley T; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Translational Research Unit, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany., Klingmüller U; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Division Systems Biology of Signal Transduction, German Cancer Research Center (DKFZ), Heidelberg, Germany., Schulze A; Division of Tumor Metabolism and Microenvironment, German Cancer Research Center (DKFZ), Heidelberg, Germany., Thomas M; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Department of Oncology, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany., Christopoulos P; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Department of Oncology, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany., Sültmann H; Division of Cancer Genome Research, German Cancer Research Center (DKFZ), German Cancer Consortium (DKTK), National Center for Tumor Diseases (NCT), Im Neuenheimer Feld 280, 69120, Heidelberg, Germany. h.sueltmann@dkfz.de.; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany. h.sueltmann@dkfz.de.
Source: Cancer & metabolism [Cancer Metab] 2025 Jun 16; Vol. 13 (1), pp. 28. Date of Electronic Publication: 2025 Jun 16.
Publication Type: Journal Article
Journal Info: Publisher: BioMed Central Country of Publication: England NLM ID: 101607582 Publication Model: Electronic Cited Medium: Print ISSN: 2049-3002 (Print) Linking ISSN: 20493002 NLM ISO Abbreviation: Cancer Metab Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
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  Data: Cancer-associated fibroblasts promote drug resistance in ALK-driven lung adenocarcinoma cells by upregulating lipid biosynthesis.
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  Data: <searchLink fieldCode="AU" term="%22Daum+AK%22">Daum AK</searchLink>; Division of Cancer Genome Research, German Cancer Research Center (DKFZ), German Cancer Consortium (DKTK), National Center for Tumor Diseases (NCT), Im Neuenheimer Feld 280, 69120, Heidelberg, Germany.; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Medical Faculty, Heidelberg University, Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Schlicker+L%22">Schlicker L</searchLink>; Division of Tumor Metabolism and Microenvironment, German Cancer Research Center (DKFZ), Heidelberg, Germany.; Proteomics Core Facility, German Cancer Research Center (DKFZ), Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Schneider+MA%22">Schneider MA</searchLink>; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Translational Research Unit, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Muley+T%22">Muley T</searchLink>; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Translational Research Unit, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Klingmüller+U%22">Klingmüller U</searchLink>; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Division Systems Biology of Signal Transduction, German Cancer Research Center (DKFZ), Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Schulze+A%22">Schulze A</searchLink>; Division of Tumor Metabolism and Microenvironment, German Cancer Research Center (DKFZ), Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Thomas+M%22">Thomas M</searchLink>; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Department of Oncology, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Christopoulos+P%22">Christopoulos P</searchLink>; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany.; Department of Oncology, Thoraxklinik at Heidelberg University Hospital, Heidelberg, Germany.<br /><searchLink fieldCode="AU" term="%22Sültmann+H%22">Sültmann H</searchLink>; Division of Cancer Genome Research, German Cancer Research Center (DKFZ), German Cancer Consortium (DKTK), National Center for Tumor Diseases (NCT), Im Neuenheimer Feld 280, 69120, Heidelberg, Germany. h.sueltmann@dkfz.de.; German Center for Lung Research (DZL), TLRC Heidelberg, Heidelberg, Germany. h.sueltmann@dkfz.de.
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  Data: <searchLink fieldCode="JN" term="%22101607582%22">Cancer & metabolism</searchLink> [Cancer Metab] 2025 Jun 16; Vol. 13 (1), pp. 28. <i>Date of Electronic Publication: </i>2025 Jun 16.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22BioMed+Central%22">BioMed Central </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101607582 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Print <i>ISSN: </i>2049-3002 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2220493002%22">20493002 </searchLink><i>NLM ISO Abbreviation: </i>Cancer Metab <i>Subsets: </i>PubMed not MEDLINE
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              Text: 2025 Jun 16
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