β-aminopropionitrile-induced thoracic aortopathy is refractory to cilostazol and sildenafil in mice.
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| Title: | β-aminopropionitrile-induced thoracic aortopathy is refractory to cilostazol and sildenafil in mice. |
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| Authors: | Tyagi SC; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Physiology, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Surgery, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Ito S; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Hubbuch JC; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Physiology, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Surgery, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Franklin MK; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Howatt DA; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Lu HS; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Physiology, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Daugherty A; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Physiology, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America., Sawada H; Saha Cardiovascular Research Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Saha Aortic Center, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America.; Department of Physiology, College of Medicine, University of Kentucky, Lexington, Kentucky, United States of America. |
| Source: | PloS one [PLoS One] 2025 Aug 29; Vol. 20 (8), pp. e0322434. Date of Electronic Publication: 2025 Aug 29 (Print Publication: 2025). |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Public Library of Science Country of Publication: United States NLM ID: 101285081 Publication Model: eCollection Cited Medium: Internet ISSN: 1932-6203 (Electronic) Linking ISSN: 19326203 NLM ISO Abbreviation: PLoS One Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 1932-6203 |
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| DOI: | 10.1371/journal.pone.0322434 |
